https://www.selleckchem.com/products/ab928.html
Mechanically, circCRKL could positively regulate KLF5 expression by sponging miR-141. In addition, circCRKL upregulation could hinder PCa tumor growth in vivo. These findings revealed that circCRKL inhibited the progression of PCa through upregulating KLF5 expression by sponging miR-141, elucidating a novel regulatory pathway in PCa cells. Our research suggested an underlying circRNA-targeted therapy for PCa. These findings revealed that circCRKL inhibited the progression of PCa through upregulating KLF5 expression by sponging miR-141, el
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