https://www.selleckchem.com/pr....oducts/ziritaxestat.
The tenant of this hypothesis is that loss of tolerance would not occur if the zonulin-dependent intestinal barrier function is restored, thereby preventing the influence of environmental triggers in individuals genetically susceptible to autoimmunity. This review outlines the current research and a structured hypothesis on how a dysregulated small intestinal epithelial barrier, a "leaky gut", may be important in the pathogenesis of autoimmunity in certain individuals at risk of both T1D and celiac disease. This article is protecte
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