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The steroid hormone glucocorticoid (GC) is a well-known immunosuppressant that controls T-cell-mediated adaptive immune response. In this work, we have developed a minimal kinetic network model of T-cell regulation connecting relevant experimental and clinical studies to quantitatively understand the long-term effects of GC on pro-inflammatory T-cell (T_pro) and anti-inflammatory T-cell (T_anti) dynamics. Due to the antagonistic relation between these two types of T cells, their long-term steady-state population ratio helps us to characteri
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