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001), but none received TPN. Despite earlier feeding, Kenyan patients displayed a higher prevalence of wound infections (70.8% vs 17.1%, p0.001) and sepsis (43.9% vs 4.8%, p0.001). In Kenya, survivors and non survivors displayed no difference in sterile bag use, hemodynamic stability, all cause infection rates, or antibiotic free hospital days. Defect closure (p0.001) and enteral feeding initiation (p0.001) were most predictive of survival. Improving immediate response strategies for gastroschisis in Kenya could improve su
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