https://www.selleckchem.com/pr....oducts/gsk1120212-jt
05). However, inhibition of MEG3 in WiT49 cells significantly promoted cell growth and metastasis compared with cells in negative control group (p0.05). In addition, MEG3 influenced the protein expression of β-catenin by regulating the Wnt/β-catenin pathway. MEG3 was low-expressed in WT tissues and blood samples. Meanwhile, it could inhibit the proliferation and metastasis of WT cells via wt/β-catenin pathways. All our findings indicated that MEG3 served as a potential target for the diagnosis, treatment and prognosis p
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