https://www.selleckchem.com/pr....oducts/salinosporami
Mechanistic studies indicated that mBMPR1A-mFc administration promoted osteoblastogenesis by activating Wnt/Lrp5/β-catenin signaling while decreasing osteoclastogenesis by inhibiting the RANKL/RANK/OPG pathway. Our novel findings provide solid evidence for the application of mBMPR1A-mFc as a therapeutic treatment for radiation-induced osteoporosis. AJTR Copyright © 2020.Oxidative stress can trigger DNA damage response and activation of cellular senescence. Accumulating studies have demonstrated that senescen
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