https://nf-kb-inhibitors.com/r....eproduction-associat
From a forward hereditary display for changed PD transport, we discovered that the conserved eukaryotic glucose-TOR (TARGET OF RAPAMYCIN) metabolic signaling community limits PD transport in leaves. Hereditary approaches and substance or physiological treatments to either improve or disrupt TOR activity demonstrate that glucose-activated TOR decreases PD transport in leaves. We further found that TOR is a lot more active in mature leaves photosynthesizing excess sugars compared to young
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