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Afterwards, we provide an update about the part of LC-associated features when it comes to pathophysiology of schizophrenia, emphasizing the cognitive and inspirational deficits. We suggest that schizophrenia phenomenology, in specific cognitive signs, might be explained by an abnormal interacting with each other between genetic susceptibility and stress-initiated LC-NE disorder. This in turn, contributes to imbalance between LC task settings, dysfunctional legislation of brain communi
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