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These actions were linked to modulation of the redox-sensitive transcription factor JunB and impairment of the mitochondrial respiratory system. Our results indicate that pharmacological stimulation of the α7nAchR could be a promising target for treatment of patients with skin fibrotic diseases. Moreover, our results suggest a mechanistic axis of collagen synthesis regulation via the mitochondrial respiratory system. Evidence indicates that patterns of biological reactivity underlie different forms of aggression, but greater precision is needed in research
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