https://www.selleckchem.com/products/AZD0530.html
felis colonization compared to infected TGF-β mice. In a T cell-deficient background using TGF-β xRag1KO mice, H. felis colonization was significantly lower when DC-derived TGF-β was absent, revealing a direct, innate function of DC in controlling H. felis infection independent of Treg induction. Our findings indicate that DC-derived TGF-β mediates Helicobacter-induced Treg response and attenuates the inflammatory Th1 response. We also demonstrated a previously unrecognized innate role of DC controlling Helicobacter colonization via a T
Like
Comment
Share
