https://www.selleckchem.com/pr....oducts/Sodium-orthov
Together, these data indicate that the TUBA1A-rich microtubule tracks that are assembled during development are essential for mature neuron function and maintenance of synapses over time.Significance Statement Defects in axonal trafficking have been reported in models of movement disorders, but it has been unclear if trafficking defects are coincident or causative. Our results suggest that deficits in TUBA1A αTubulin can cause trafficking defects that impair synaptic maintenance leading to a movement disorder without axon d
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