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These conclusions indicate that adipose tissue itself plays a critical role when you look at the pathophysiology of OA. Susceptibility to posttraumatic OA was reintroduced into LD mice making use of implantation of a small adipose tissue depot derived from wild-type animals or mouse embryonic fibroblasts that undergo natural adipogenesis, implicating paracrine signaling from fat, instead of weight, as a mediator of combined degeneration.This knowledge article discusses
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