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In addition, the cellular demise was improved by co-expression of ALG-2 and ESCRT-I, indicating that ALG-2 likely promotes CDIP1-induced cellular death by advertising the organization between CDIP1 and ESCRT-I. We additionally found that CDIP1 binds to vesicle-associated membrane layer protein-associated protein (VAP)A and VAPB through the two phenylalanines in an acidic tract (FFAT)-like theme in the C-terminal region of CDIP1, mutations of which r
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