https://www.selleckchem.com/products/GDC-0449.html
Necroptosis deficient mice had increased survival and reduced weight loss post-IAV infection, as well as increased antioxidant and mitochondrial function, indicating partial protection to IAV infection. These findings were confirmed in vitro by pre-treatment of human and rat myocytes with antioxidants or necroptosis inhibitors, which blunted oxidative stress and mitochondrial damage after IAV infection. Conclusions This study provides the first evidence that the cardiac proteome and phosphoproteome are significantly altered post pulmon
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