https://www.selleckchem.com/pr....oducts/vevorisertib-
We reported that the absence of a guanine oxidized (GO) system or the AP-endonucleases Nfo, ExoA and Nth promoted stress-associated mutagenesis (SAM) in B. subtilis YB955 (hisC952, metB5, leuc427). Moreover, MutY-promoted SAM was Mfd-dependent suggesting that transcriptional transactions over non-bulky DNA lesions promoted error-prone repair. Here, we inquired whether Mfd and GreA, which control transcription-coupled repair and transcription fidelity, influence the mutagenic events occurring in nutritionally stress
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