https://www.selleckchem.com/products/sbi-477.html
With early diagnosis, colorectal cancer (CRC) is a curable disease. As studies in the past 15 years have shown, specific genetic changes occur in the neoplastic transformation of normal colonic epithelium to benign adenoma until becoming adenocarcinoma. Considering that dynamic, we aimed to determine how v-raf murine sarcoma viral oncogene homolog B1 and Kirsten rat sarcoma mutations relate to the location, histopathology, and degree of tumor differentiation in CRC. With a cross-sectional design involving an observational analytical app
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