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Functional experiments determined that reduction of HNF1A-AS1 or marketing of miR-124 inhibited cell migration and invasion in addition to glycolysis in CRC cells. What' more, luciferase reporter assay manifested that miR-124 was a target of HNF1A-AS1 and MYO6 had been a target mRNA of miR-124 in CRC cells. Furthermore, reverse experiments showed that the results of si-HNF1A-AS1 on colorectal disease cells were impaired by anti-miR-124 and also the aftereffects of large mi