https://www.selleckchem.com/pr....oducts/ci994-tacedin
Importantly, the increases in α-syn expression and neurotoxicity were relieved by Parkin overexpression. CONCLUSIONS By establishing stable cell lines and animal models that overexpress Parkin, we confirmed Parkin as an important factor in METH-induced α-syn degradation dysfunction in vitro and in vivo. Parkin may be a promising target for the treatment of METH-induced neurotoxicity. © 2020 The Authors. Brain and Behavior published by Wiley Periodicals, Inc.BACKGROUND Within the last decade, advances and availability in radio
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