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No ophthalmologic changes from standard or ocular all-causality TEAEs required permanent treatment discontinuation.Interleukin-31 (IL-31) is a significant protein tangled up in severe inflammatory epidermis problems. Its signaling pathway is mediated through two type I cytokine receptors, IL-31RA (also known as the gp130-like receptor) additionally the oncostatin M receptor (OSMR). Understanding molecular details within these communications is helpful for building antagonist anti-IL-31 monoclonal an
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