https://www.selleckchem.com/products/ox04528.html
The local administration of CCL22 in the kidney facilitated Treg accumulation and reduced glomerular crescent formation. This study revealed a new mechanism whereby effector Tregs migrate into the inflammatory kidney the CCL22/17-CCR4 axis that is facilitated by M2-like type macrophages that are induced by IL-6R blockade. This study revealed a new mechanism whereby effector Tregs migrate into the inflammatory kidney via the CCL22/17-CCR4 axis that is facilitated by M2-like type macrophages that are induced by IL-6R blockade.In various t
Like
Comment
Share
