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015 and p  .001, respectively), but these associations were perturbed by liraglutide. In a physiologically relevant setting that mirrored regular feeding in subjects with T2D, liraglutide promoted potentially beneficial changes on postprandial apoB48 metabolism. Using our data in an integrated metabolic model, we describe how the action of liraglutide in T2D on chylomicron and VLDL kinetics could lead to decreased generation of remnant lipoproteins. In a physiologically relevant setting that mirrored regular feeding in subjects wit

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