https://isoxazole9antagonist.c....om/cisplatin-loaded-
Here, we investigate the contribution associated with inositol-requiring protein-1α-X-box binding protein-1 (XBP1)-mediated UPR signaling pathway into the pathogenesis of spinal-cord injury (SCI). We prove that removal of Xbp1 caused an exacerbated ATF4/CHOP signaling in cultured mouse oligodendrocyte (OL) progenitor cells and enhanced their sensitivity to ER anxiety. Similar results had been also seen using the Xbp1 pathway inhibitor toyocamycin. Also, OL lineage-speci
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