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Hepcidin also played a task in reducing α-syn accumulation in rotenone and α-syn overexpression conditions. We identified that the possible clearance effect of hepcidin on α-syn had been mediated by the autophagy pathway making use of pretreatment with autophagy inhibitors (3-MA and CQ) and detection of autophagy protein markers (LC3II/I and p62). In conclusion, hepcidin eliminated α-syn phrase via the autophagy path in rotenone-treated and
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