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xicam at the concentration of 100 μmol/L significantly promoted the mineralization of LPS-hDPCs (P less then 0.05). CONCLUSION In this study, meloxicam promoted the proliferation of hDPCs, inhibited the inflammatory reaction and promoted differentiation and mineralization of hDPCs under LPS irritation. The present results suggest that meloxicam may play a role in anti-inflammation and repair of pulp inflammation.OBJECTIVE To investigate the expression changes of the epigenetic regulator enhancer of zeste homolog 2 (EZH2) during pulp infl
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