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Consequently, double luciferase reporter gene assay demonstrated that Beclin-1 had been a primary target of miR-506-3p, and reintroduction of Beclin-1 could antagonize the suppressive effect of miR-506-3p overexpression on fibroblast proliferation, autophagy, therefore the intermolecular cross-links in collagen synthesis. Taken collectively, our results indicated that miR-506-3p regulated autophagy and proliferation in post-burn epidermis fibroblasts through post-tran
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