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59 ± 1.65 °C (CEM43 = 447 ± 79), respectively. FUS improves the effect of RT significantly by reducing metabolic activity in T98G cells 48 h (RT 96.47 ± 8.29%; FUS+RT 79.38 ± 14.93%; p = 0.012) and in PC-3cells 72 h (54.20 ± 10.85%; 41.01 ± 11.17%; p = 0.016) after therapy, but not in FaDu cells. Mechanistically, FUS+RT leads to increased apoptosis and enhancement of DNA double-strand breaks compared to RT alone in T98G and PC-3cells. Our in vitro findings demonstrate that FUS has good potential to sensitize glioblastoma and prostate ca
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