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This study increases our comprehension of exactly how mitochondria modulate neuronal homeostasis and personal behavior under physiopathological conditions. Influenza A virus (IAV) is a lytic RNA virus that creates receptor-interacting serine/threonine-protein kinase 3 (RIPK3)-mediated pathways of apoptosis and combined lineage kinase domain-like pseudokinase (MLKL)-dependent necroptosis in contaminated cells. ZBP1 initiates RIPK3-driven cellular demise by sensing IAV RNA and acti
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