https://www.selleckchem.com/pr....oducts/bms-265246.ht
The local administration of CCL22 in the kidney facilitated Treg accumulation and reduced glomerular crescent formation. This study revealed a new mechanism whereby effector Tregs migrate into the inflammatory kidney the CCL22/17-CCR4 axis that is facilitated by M2-like type macrophages that are induced by IL-6R blockade. This study revealed a new mechanism whereby effector Tregs migrate into the inflammatory kidney via the CCL22/17-CCR4 axis that is facilitated by M2-like type macrophages that are induced by IL-6R blockade.In variou
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