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Inborn resistant memory is defined by enhanced release of pro-inflammatory cytokines by inborn protected cells following a secondary challenge with pattern recognition receptor (PRR) ligands. Peripheral bloodstream monocytes isolated from 33 customers with intermediate- or high-risk NMIBC pre and post two or five induction BCG instillations were activated because of the PRR ligand lipopolysaccharide (LPS). Inflammatory cytokine levels into the trad
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