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gies required to mitigate the negative effects of EDC and environmental toxicant exposure on human fertility and fecundity.Many studies have reported that PM2.5 was associated with mortality, but these were criticized for unmeasured confounding, not using causal modeling, and not focusing on changes in exposure and mortality rates. Recent studies have used propensity scores, a causal modeling approach that requires the assumption of no unmeasured confounders. We used differences in differences, a causal modeling approach that focuses on exposure changes, an
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