https://www.selleckchem.com/products/neo2734.html
miR-132 quantity was reduced in Aβ25-35-challenged neuronal cells, and negatively controlled by SOX21-AS1. miR-132 knockdown abolished the effect of SOX21-AS1 silencing on Aβ25-35-induced neuronal damage, indicating that SOX21-AS1 controls Aβ25-35-induced neuronal damage via regulating miR-132. The PI3K/AKT signaling was repressed in Aβ25-35-challenged cells, but this effect was counteracted upon overexpression of miR-132. In conclusion, SOX21-AS1 knockdown mitigated Aβ25-35-dependent neuronal cell damage by promoting miR-132/PI3K/AKT p
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