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Both P- and AF-WPS breathing induced a rise in systolic hypertension, thrombogenicity and plasma concentration of fibrinogen and von Willebrand factor. In heart homogenates, AF-WPS breathing caused an increase of 8-isoprostane and a decrease in the quantities of reduced glutathione (GSH) and superoxide dismutase (SOD). Nonetheless, P-WPS reduced just the activity of SOD. The levels of tumour necrosis factor α and interleukin 1β were increased only in heart homogenates of mic
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