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CONCLUSIONS Our results shed light on the molecular framework that may underlie extra-choroidal CSF production and we propose that AQP1 and NKCC1 within the leptomeningeal vasculature, specifically at the capillary level, are poised to play a role in CSF production throughout the central nervous system.BACKGROUND When exposed to a novel dynamic perturbation, participants adapt by changing their movements' dynamics. This adaptation is achieved by constructing an internal representation of the perturbation, which allows for applying force