https://www.selleckchem.com/pr....oducts/SB-203580.htm
expression and acetylation. However, miR-874-3p inhibitor did not further decrease p53 acetylation and expression in cell overexpressing HDAC1. Collectively, the results of the present study indicated that miR-874-3p was upregulated in PCOS and promoted testosterone-induced GC apoptosis by suppressing HDAC1-mediated p53 deacetylation. Therefore, the present study improved the current understanding of the pathogenesis of PCOS and GC apoptosis.Νon-small cell lung cancer (NSCLC) is the most frequently diagnosed type of cancer, and the mo
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