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Results showed that XIST expression was increased but miR-1264 appearance amount ended up being reduced in the serum of AAA clients. XIST exhaustion hampered man aorta VSMCs (HA-VSMCs' power to proliferate and stimulate apoptosis, while repressing miR-1264 appearance through an unmediated conversation. Additionally, the influence of XIST knockdown on apoptosis and proliferation could be rescued by an miR-1264 inhibitor. Subsequent molecular investigations indicated that WNT5A was miR-1264's target
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