https://www.selleckchem.com/products/t-5224.html
Mechanistically, SNHG1 directly targeted miR-376a, and CBS was a target of miR-376a. Moreover, SNHG1 exert its function via inhibiting miR-376a to regulate CBS expression.Conclusion LncRNA SNHG1 depressed apoptosis and inflammation of IS cell model via inhibiting miR-376a and upregulating CBS/H2S signal. These results show light on underlying mechanisms of IS and provide potential targets for IS therapy.Background Tobacco use is recognized as a form of addiction and remains a significant health concern. Despite this well accepted problem
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