https://duvelisibinhibitor.com..../position-of-ordinar
The in-patient EHDs had been found to consider the conventional homeodomain fold. Most importantly, the base-specific communications within the major groove required for the affinity/specificity of wild-type EHD had been preserved in (EHD[R53A])2. Bacterial assays verified that the base-specific communications are retained under cellular problems. These observations suggest that the R53A mutation just causes a loss of the arginine-phosphate interaction at the protein-DNA user interface, which reduces t
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