https://www.selleckchem.com/MEK.html
aureus, purR mutants have enhanced invasion of these non-professional phagocytes, consistent with the requirement of FnBPs for invasion of these cells. This correlates with purR mutants having both increased transcription of fnb genes, resulting in higher levels of surface-exposed FnBPs to promote invasion. These data provide important contributions to our understanding of how the pathogenesis of S. aureus is affected by sensing of purine levels during infection of the mammalian host. Copyright © 2020 Goncheva et al.Chronic bacterial infections are
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