https://www.selleckchem.com/pr....oducts/guanidine-thi
CP administration significantly increases KIM-1, NGAL, Cr, BUN, MDA, NO, IL-1β, and TNF-α level. It also decreases GSH concentration, SOD, GPx, and CAT function. Pretreatment with GA prevented these changes. Histopathological assessments approved the GA protective effect. Our results showed that GA is possibly effective as a protective agent in cyclophosphamide-associated toxicities. Our results showed that GA is possibly effective as a protective agent in cyclophosphamide-associated toxicities. Aim of the present study is
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