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We evaluated the part of SOX2-OT/SOX2 and SOX2-OT/SOX2/GLI-1 axes using RT-qPCR, western blot, immunofluorescence analyses, gene silencing, cellular cytotoxic, and ChIP-qPCR assays on real human cellular outlines, solid lung malignant tumors, and regular lung structure. We detected that the SOX2-OT/SOX2/GLI-1 axis encourages resistance to tyrosine kinase inhibitor (TKI)-erlotinib and cisplatin-based therapy . Proof using this study program that SOX2-OT modulates the
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