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Further investigation showed that the activation of RIG-I was largely responsible for poly(dAdT)-mediated HSV-2 inhibition and IFN/ISGs induction in the cervical epithelial cells, as RIG-I knockout abolished the poly(dAdT) actions. These observations demonstrate the importance for design and development of AT-rich dsDNA-based intervention strategies to control HSV-2 mucosal transmission in FRT.The intestinal microbiota is a critical component of mucosal health as evidenced by the fact that alterations in the taxonomic composition of th
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