https://www.selleckchem.com/pe....ptide/gp91ds-tat.htm
01), lipogenesis (p ˂ 0.05) and lipolysis (p ˂ 0.05) despite an increase in tissue weight. The results provide evidence that impaired hepatic triglycerides and dysregulation of some CYP450 proteins may have been involved in the development of hepatic steatosis. The low metabolic activity of brown adipose tissue may have contributed to visceral adiposity as well as triglyceride accumulation during the postmenopausal period.The SOS response is induced upon DNA damage and the inhibition of Z ring formation by the product of the sulA gene
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