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SDC2 had been highly-expressed, while miR-9 was poorly-expressed in atherosclerosis. Furthermore, miR-9 focused SDC2 and negatively-regulated its expression. Up-regulation of miR-9 reduced aortic plaque location, the proliferation of collagen fibers, Mac-3-labeled macrophages and amounts of IL-6, IL-1β, and TNF-α by curbing SDC2 together with FAK/ERK signaling pathway, thereby ameliorating atherosclerosis in ACS mice. In closing, the current study provides evidence that miR-9 retards atheroscle
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