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g., ROS and NO), decreased the level of GSH, and improved oxidative DNA damage and lipid peroxidation in cells, that have been effectively reversed by celastrol treatment. Additionally, inflammatory reactions caused by gamma irradiation, as demonstrated by enhanced quantities of IL-6, TNF-α, and IL-1β, were additionally blocked by celastrol. The increased activity of NF-κB DNA binding following gamma radiation was significantly attenuated after celastrol treatment. In the irr
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