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The phrase of Traf6, MyD88, and Tnf in Brl-3A cells and therefore of MYD88, TNF, and IL1B in L-02 cells were substantially upregulated by PCB52. Regularly, overexpression of TLR4, MyD88, Traf6, and NF-κB p65 proteins ended up being seen in PCB52-treated cells, showing activation of inflammatory responses. Nevertheless, no alterations in kelch-like ECH-associated necessary protein 1 (keap1), nuclear factor-erythroid 2-related aspect (nrf2), and heme oxygenase-1 proteins were notice
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