https://www.selleckchem.com/products/ABT-263.html
However, these parameters were not significantly different when comparing WT and EC-IFT88 mice after DOX treatment. The loss of EC primary cilia accelerated DOX-mediated mortality and reduced cardiac function, suggesting pathways downstream of ciliary-mediated signal transduction as potential targets to promote EC support of cardiomyocyte function during DOX treatment. The loss of EC primary cilia accelerated DOX-mediated mortality and reduced cardiac function, suggesting pathways downstream of ciliary-mediated signal transduction as po
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