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In the current research we show that the partly unfolded very first Type III domain of fibronectin, III-1c, activates a toll-receptor/NF-κB pathway ultimately causing an increase in the expression of IL-8. Utilizing a 3-D style of tumor-associated extracellular matrix, we demonstrate that lung cancer cells seeded onto this matrix activate a TLR4/NF-κB signaling pathway resulting in a robust escalation in the production of IL-8. Cytokine
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