https://www.selleckchem.com/
Taken together, these findings suggest that swainsonine along with calystegines, are probably responsible for the activation of glial cells due to a possible lysosomal dysfunction and therefore intracellular storage. Our results demonstrate that this in vitro glial cell model is a very good alternative to in vivo studies that require several weeks of animal intoxication to observe similar neurotoxic effects. Accumulating evidence links fine particulate matter (PM ) to premature mortality, cardiovascular disease, and respiratory disease. However, less is kno
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