https://www.selleckchem.com/products/tpca-1.html
Compared to those from healthy controls, Tfh2 cells from AR patients had a greater capacity to induce CD23 expression on switched memory B cells via IL-4, which was unable to be sufficiently suppressed by AR-associated Tfr cells with defective IL-10 expression. CD23 expression on switched memory B cells was downregulated after 12-month AIT, which positively associated with disease remission in AR patients. CONCLUSION T-B cell interaction, bridged by CD23 expression particularly on switched memory B cells, may be involved in the disease p
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