https://ym155inhibitor.com/prm....t6-assists-the-oncog
BM-MSCs additionally favored the histological structure data recovery and proliferative ability of pancreatic islet cells. Moreover, BM-MSC management effectively reversed the abnormal expression of insulin resistance-related proteins including GLUT4, phosphorylated insulin receptor substrate 1, and protein kinase Akt and proinflammatory cytokines IL-6 and TNFα in liver. These results suggested that MSCs transplantation during hyperinsulinemia could prevent many prospectiv
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